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When stimulated by APC inactivation, Wnt signaling synergizes retinoblastoma (Rb) inactivation to induce apoptosis through a mechanism mediated by increased activity of the target of rapamycin complex 1 (TORC1), causing metabolic, oxidative, and energy stress [144]
Bondo
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Cetinel, S., Ercan, F., Cikler, E., Contuk, G
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